MD University of Milan, Italy, 1976
PhD. Scuola Normale Superiore, Pisa, Italy 1982
Post-Doc Columbia University, Ctr. for Neurobiology and Behaviour 1983-87
I have been teaching “Memory and Learning” at UBC since 2012.
Previously, I taught a section of pharmacology for medical students at UT Southwestern in Dallas (1987-96) and a section of physiology for medical residents at the University of Pisa (1981-83).
I have a long-standing interest in understanding how social relationships and culture shape the human brain, and how the brain in turn orchestrates our behaviours, with a focus on psychiatry.
Peer-reviewed journals (selection):
Hildebrand, M.E., Smith, P., Bladen, C., Eduljee, C., Xie J.Y., Chen, L., Fee-Maki, M., Doering, C., Mezeyova, J., Xhu, Y., Belardetti, F., Pajouhesh, H., Parker, D., Arneric, S.P., Parmar, M., Porreca, F., Tringham, E., Zamponi, G., Snutch, T. A novel slow-inactivation-specific ion channel modulator attenuates neuropathic pain. Pain, 152: 833-843, 2011.
Bolshakov, V., Carboni, L., Cobb, M.H., Siegelbaum, S.A. and Belardetti, F. Dual MAP kinase pathways mediate opposing forms of long-term plasticity at CA3-CA1 synapses. Nature Neurosci. 3: 1107-12, 2000.
Wilk-Blaszczak, M.A., Stein, B., Xu, S. Barbosa, M.S., Cobb, M.H., and Belardetti, F. The Mitogen-Activated Protein Kinase p38-2 Is Necessary for the Inhibition of N-Type Calcium Current by Bradykinin. J.Neurosci.18:112-118, 1998.
Wilk-Blaszczak, M.A., Singer, W.D., Quill, T., Miller, W., Frost, J.A., Sternweis, P.C. and Belardetti, F. The monomeric G proteins Rac1/Cdc42 are required for the inhibition of voltage-dependent calcium current by bradykinin. J.Neurosci.17:4094-4100, 1997.
Wilk-Blaszczak, M.A., Singer, W.D., Gutowski, S., Sternweis, P.C. and Belardetti F. The G protein G13 mediates bradykinin inhibition of voltage-dependent calcium current by bradykinin. Neuron 13:1215-1224, 1994.
Wilk-Blaszczak, M.A., Gutowski,S., Sternweis, P. and Belardetti, F. Bradykinin modulates potassium and calcium currents in neuroblastoma hybrid cells via different pertussis toxin-insensitive pathways. Neuron, 12:109-116, 1994.
Taussig, R., Sanchez, S., Rifo, M., Gilman, A.G. and Belardetti, F. Inhibition of the w-Conotoxin-Sensitive Calcium Current by Distinct G Proteins. Neuron, 8:799-809, 1992.
Belardetti, F., Campbell, W.B., Falck, J.R., Demontis, G. and Rosolowsky, M. Products of heme-catalyzed transformation of the arachidonate derivative, 12-HPETE, open S-type K+ channels in Aplysia. Neuron 3:497-505, 1989.
Piomelli, D., Volterra, A., Dale, A., Siegelbaum, S.A., Kandel, E.R., Schwartz, J.H. and Belardetti, F. Lipoxygenase metabolites of arachidonic acid as second messengers for presynaptic inhibition of Aplysia sensory cells. Nature 328:38-43, 1987.
Belardetti, F., Kandel, E.R. and Siegelbaum, S.A. Neuronal inhibition by the peptide FMRFamide involves opening of S-K+ channels. Nature 325:153-156, 1987.
Belardetti, F., Schacher, S., Kandel, E.R. and Siegelbaum, S.A. The growth cones of Aplysia sensory neurons: Modulation by serotonin of action potential duration and single potassium channel currents. Proc. Natl. Acad. Sci. USA 83:7094-7098, 1986.